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Get Free AccessTwo important questions regarding the genetics of pancreatic adenocarcinoma (PDAC) are 1. Which germline genetic variants influence the incidence of this cancer; and 2. Whether PDAC causally predisposes to associated non-malignant phenotypes, such as type 2 diabetes (T2D) and venous thromboembolism (VTE).
Marcus Tan, Chelsea A. Isom, Yangzi Liu, David‐Alexandre Trégouët, Sara Lindström, Lu Wang, Erin N. Smith, William Gordon, Astrid van Hylckama Vlieg, Mariza De Andrade, Jennifer A. Brody, Jack Pattee, Jeffrey Haessler, Ben Brumpton, Daniel I. Chasman, Pierre Suchon, Ming‐Huei Chen, Constance Turman, Marine Germain, Kerri L. Wiggins, James MacDonald, Sigrid K. Brækkan, Sebastian M. Armasu, Nathan Pankratz, Rabecca D Jackson, Jonas B. Nielsen, Franco Giulianini, Marja Puurunen, Manal Ibrahim, Susan R. Heckbert, Theo K. Bammler, Kelly A. Frazer, Bryan M. McCauley, Kent D. Taylor, James S. Pankow, Alexander P. Reiner, Maiken E. Gabrielsen, Jean‐François Deleuze, Christopher J. O’Donnell, Jihye Kim, Barbara McKnight, Peter Kraft, J.-B. Hansen, Frits R. Rosendaal, John A. Heit, Bruce M. Psaty, Weihong Tang, Charles Kooperberg, Kristian Hveem, Paul M. Ridker, Pierre‐Emmanuel Morange, Andrew M. Johnson, Christopher Kabrhel, David‐Alexandre Trégouët, Nicholas H. Smith, Lang Wu, Dan Zhou, Eric R. Gamazon (2024). Transcriptome-wide association study and Mendelian randomization in pancreatic cancer identifies susceptibility genes and causal relationships with type 2 diabetes and venous thromboembolism. EBioMedicine, 106, pp. 105233-105233, DOI: 10.1016/j.ebiom.2024.105233.
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Type
Article
Year
2024
Authors
58
Datasets
0
Total Files
0
Language
English
Journal
EBioMedicine
DOI
10.1016/j.ebiom.2024.105233
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