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Get Free Access(Cell Reports 15, 1481–1492; May 17, 2016) In the originally published version of this article, Michaela Lang, who contributed some of the data shown in Figure 7, was mistakenly omitted from the author list. The corrected version of the article now appears online. The authors regret the error. The Tumor Suppressor Hace1 Is a Critical Regulator of TNFR1-Mediated Cell FateTortola et al.Cell ReportsMay 5, 2016In BriefTortola et al. report that the E3 ubiquitin ligase HACE1 is a gatekeeper of TNFR1-mediated cell fate. Hace1 deficiency impairs TNF-driven NF-κB activation and apoptosis and predisposes cells to necroptosis. Consequently, hace1–/– mice show enhanced colitis and colon cancer, which can be reverted by inactivation of pro-necroptotic kinase RIP3 and TNFR1. Full-Text PDF Open Access
Luigi Tortola, Roberto Nitsch, Mathieu J.M. Bertrand, Melanie Kögler, Younes Redouane, I. Kozieradzki, Iris Uribesalgo, Lilian M. Fennell, Mads Daugaard, Helene Klug, Gerald Wirnsberger, Reiner Wimmer, Thomas Perlot, Renu Sarao, Shuan Rao, Toshikatsu Hanada, Nozomi Takahashi, Elisabeth Kernbauer, Duygu Demiröz, Michaela Lang, Giulio Superti‐Furga, Thomas Decker, Andrea Pichler, Fumiyo Ikeda, Guido Guido Kroemer, Peter Vandenabeele, Poul H. Sorensen, Josef Penninger (2016). The Tumor Suppressor Hace1 Is a Critical Regulator of TNFR1-Mediated Cell Fate. , 16 (12), DOI: https://doi.org/10.1016/j.celrep.2016.08.072.
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Type
Corrigendum
Year
2016
Authors
28
Datasets
0
Total Files
0
Language
en
DOI
https://doi.org/10.1016/j.celrep.2016.08.072
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