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  5. Spontaneous atopic dermatitis in mice with a defective skin barrier is independent of ILC2 and mediated by IL‐1β

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Article
English
2019

Spontaneous atopic dermatitis in mice with a defective skin barrier is independent of ILC2 and mediated by IL‐1β

0 Datasets

0 Files

English
2019
Allergy
Vol 74 (10)
DOI: 10.1111/all.13801

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Luke O'neill
Luke O'neill

Trinity College Dublin

Verified
Christian Schwartz
Tara Moran
Sean P. Saunders
+12 more

Abstract

Atopic dermatitis (AD) is one of the most common skin diseases with a multifactorial etiology. Mutations leading to loss of skin barrier function are associated with the development of AD with group 2 innate lymphoid cells (ILC2) promoting acute skin inflammation. Filaggrin-mutant (Flg

How to cite this publication

Christian Schwartz, Tara Moran, Sean P. Saunders, Agnieszka Kaszlikowska, Achilleas Floudas, Joana Bom, Gabriel Núñez, Yoichiro Iwakura, Luke O'neill, Alan D. Irvine, Andrew N. J. McKenzie, Graham S. Ogg, Patrick Walsh, Jocelyne Demengeot, Padraic G. Fallon (2019). Spontaneous atopic dermatitis in mice with a defective skin barrier is independent of ILC2 and mediated by IL‐1β. Allergy, 74(10), pp. 1920-1933, DOI: 10.1111/all.13801.

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Publication Details

Type

Article

Year

2019

Authors

15

Datasets

0

Total Files

0

Language

English

Journal

Allergy

DOI

10.1111/all.13801

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