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Get Free AccessPurpose: Macrophage specific knockout of PHD-2 prolylhydroxylase domain protein 2 (PHD-2-/-), the crucial cellular oxygen sensor, increased anti-inflammatory M2 macrophages, angiogenesis and arteriogenesis in mice. As macrophages and angiogenesis are known to enhance progression of atherosclerotic plaques, we hypothesized that macrophage PHD-2-/- would increase plaque size and progression.
Thomas Theelen, Elke Marsch, Jasper A. F. Demandt, Clairy Dinjens, Jack P.M. Cleutjens, M. Gijbels, Peter Carmeliet, Mat J.A.P. Daemen, Judith C. Sluimer (2014). P477Myeloid PHD-2 deficiency leads to enlarged and fibrotic atherosclerotic plaques in mice. , 103(suppl 1), DOI: https://doi.org/10.1093/cvr/cvu091.153.
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Type
Article
Year
2014
Authors
9
Datasets
0
Total Files
0
Language
en
DOI
https://doi.org/10.1093/cvr/cvu091.153
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