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Get Free AccessOverall, PHD1 knockout mice display a metabolic phenotype that generally is deemed protective for cardiovascular disease. Future studies should focus on the efficacy, safety, and gender-specific effects of PHD1 inhibition in humans, and unravel the molecular actors responsible for PHD1-driven, likely intestinal, and regulation of cholesterol metabolism.
Elke Marsch, Jasper A. F. Demandt, Thomas Theelen, Bibian M. E. Tullemans, Kristiaan Wouters, Mariëtte R. Boon, Theo H. van Dijk, Marion J. Gijbels, Ludwig J. Dubois, Steven J.R. Meex, Massimiliano Mazzone, Gene Hung, Edward A. Fisher, Erik A.L. Biessen, Mat J.A.P. Daemen, Patrick C.N. Rensen, Peter Carmeliet, Albert K. Groen, Judith C. Sluimer (2016). Deficiency of the oxygen sensor prolyl hydroxylase 1 attenuates hypercholesterolaemia, atherosclerosis, and hyperglycaemia. , 37(39), DOI: https://doi.org/10.1093/eurheartj/ehw156.
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Type
Article
Year
2016
Authors
19
Datasets
0
Total Files
0
Language
en
DOI
https://doi.org/10.1093/eurheartj/ehw156
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