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Get Free AccessKey Points Axl inhibition by BGB324 is active in FLT3-mutated and FLT3 wild-type AML, and presence of Axl and Gas6 are required for therapeutic efficacy. AML cells educate BMDSCs to secrete Gas6, which mediates leukemia cell proliferation and therapy resistance.
Isabel Ben‐Batalla, Alexander Schultze, Mark Wroblewski, Robert Erdmann, Michael Heuser, Jonas Waizenegger, Kristoffer Riecken, Mascha Binder, Denis M. Schewe, Stefanie Sawall, Victoria Witzke, Miguel Cubas‐Cordova, Melanie Janning, Jasmin Wellbrock, Boris Fehse, Christian Hagel, Jürgen Krauter, Arnold Ganser, James B. Lorens, Walter Fiedler, Peter Carmeliet, Klaus Pantel, Carsten Bokemeyer, Sonja Loges (2013). Axl, a prognostic and therapeutic target in acute myeloid leukemia mediates paracrine crosstalk of leukemia cells with bone marrow stroma. , 122(14), DOI: https://doi.org/10.1182/blood-2013-03-491431.
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Type
Article
Year
2013
Authors
24
Datasets
0
Total Files
0
Language
en
DOI
https://doi.org/10.1182/blood-2013-03-491431
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