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Get Free AccessWhen the orthologue of tumor suppressor protein p53 (TP53), cep-1, is inactivated in Caenorhabditis elegans, the nematodes manifest an autophagy-dependent increase in lifespan. A recent paper by Yang et al. demonstrates that accelerated aging phenotype of autophagy-deficient mice can be reduced by the knockout (KO) of Trp53. These findings point to a complex bidirectional crosstalk between autophagy and TP53 that has vast implications for the aging process.
Valentina Sica, Guido Guido Kroemer (2020). A bidirectional crosstalk between autophagy and TP53 determines the pace of aging. , 7(5), DOI: https://doi.org/10.1080/23723556.2020.1769434.
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Type
Article
Year
2020
Authors
2
Datasets
0
Total Files
0
Language
en
DOI
https://doi.org/10.1080/23723556.2020.1769434
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